Answer by Steven Fowkes:
Bill Skaggs is right re early diagnosis. And also because the underlying causes are not understood. What would you look for? Some are even questioning that MS is fundamentally autoimmune. Look up “venous insufficiency and MS.”
But there are a hundred other diagnoses that would be consistent with your symptoms. One would be vasospasm. Magnesium deficiency could cause temporary contraction of smooth muscles in an artery somewhere, which could restrict blood flow to specific nerves and cause a temporary weakness and brief neuropathy. Like when you lie in the wrong position and your leg or arm “goes to sleep.” And when it wakes up, pins and needles.
If you have some other reason to be concerned about MS specifically, one factor that you could measure that seems to be connected to the development of MS is low body temperature. Actually it is low basal metabolic rate, but body temperature is much easier to measure. Is your body temperature often more than a degree below normal? Do you have cold hands and feet? Do you have trouble warming up after getting chilled? Do you have a tendency to suffer constipation? Are you depressed? Low basal metabolic rate is associated with autoimmune diseases. This is something that you could do something about.
MS in particular has a sex-skewed profile, affecting women when they are young and men when they are older. I think this has to do with estrogen dominance and its suppressing effects on basal metabolic rate. Some women are constitutionally estrogen dominant, and older men with inflammation convert more testosterone to estradiol, and androstenedione to estrone. Your doctor could measure your hormones, or you could do it with saliva on an over-the-counter basis.
If you have high inflammation and induction of aromatase, your progesterone and pregnenolone may be abnormally low. Especially if you are taking a statin, which lowers cholesterol and its conversion to pregnenolone. Pregnenolone and progesterone are highly neuroprotective, to such an extent that human neurons do not survive in cultures without one or the other (or ten time more DHEA).
I’m a big fan of self-care testing modalities. I even run a self-care working group under Meetup dot com. My BHT Book presents many self-care options for treating herpes, and other viruses. And now discusses ebola strategies. Much of my blogging on Project Wellbeing is about self care. I am convinced that self-care options greatly increase the odds of actually reverse-engineering disease processes and resolving them, as opposed to the symptomatic treatments and disease-management strategy of allopathic medicine. It’s not the “safe” path by conventional thinking, which is to delegate medical decisions to “experts.” But I have found it more effective in the long run to “hack” disease instead of coexist with it.
Both pregnenolone and progesterone are sold over the counter in the USA. Pregnenolone is taken orally (the liver does not trash it efficiently). Progesterone is usually taken transdermally, in cream. You might consider exploiting this and applying progesterone cream to only one side of your body, where your symptoms are strongest, to see if you generate a differential effect. If you do, then the questions arise (1) Why is your steroidogenesis inhibited? Or (2) what kind of neurotoxic influence is at play? Mold? Heavy metals? Pesticides? Phytochemicals (from brassica?)?
Or back to the first thing I suggested, neurological brown out (hypometabolism).